NAD+ and NADH for longevity (anti-aging elixir)

Who wants to get old? No one. What do we do against aging? Nothing. Why, can you do something about it? Very much so –  based on latest evidence-based science. One of the lesser known methods is to increase the function and number of mitochondria. NAD+ has been proven to renew existing stem cells, repair damaged DNA and extend life.

What is NAD+? Nicotinamide Adenine Dinucleotide

NAD+, or nicotinamide adenine dinucleotide, is a critical coenzyme found in every cell in your body, and it’s involved in over 500 of metabolic processes. But NAD+ levels decline as we age. NAD+ has two general sets of reactions in the human body: helping turn nutrients into energy as a key player in metabolism and working as a helper molecule for proteins that regulate other cellular functions. These processes are incredibly important.

Next Generation Anti-Aging Elixir

NAD+ and NADH for longevity (anti-aging elixir)The active ingredients in  NAD+ Gold™:

  • May be your next generation, liposomal age management elixir
  • Transport NMN directly into all cells to rapidly-produce NAD+
  • Supports the replenishment of NAD+ with a unique blend of NMN and methyl donor TMG
  • The only liposomal source of NMN
  • NAD+ Gold™ can help reinforce the supply of the body’s age management molecule NAD+.

Unique new-generation elixir integrates NMN, the only direct and stable precursor to NAD+. Along with, the methyl donor TMG to fully support balanced and continuous NAD+ metabolism. Besides that, it can support replenishing and rejuvenating the cells and metabolism. A true game-changer of how to age. Another booster of mental and physical health is essential oils – like doTERRA.

What helps mitochondria function?

Everything involved in the Krebs cycle, protects mitochondria from oxidative damage and is considered a nutritional supplement to mitochondria. These can only be obtained with average nutrition in uncertain quantities and possibly.

Vitamins B: Not only is vitamin B3 known to be an advantage of NAD, but virtually all vitamins in the B group are important for mitochondrial function (Depeint et al., 2006).

Vitamin D: Vitamin D levels are known to be closely related to muscle strength, which is due to the fact that vitamin D stimulates mitochondrial formation and activity (Ryan et al., 2016), while protecting mitochondria from already harmful, increased oxygen utilization (Ricca et al., 2018).

Acetyl L-Carnitine: Part of the mitochondrial inner membrane, stimulates mitochondrial function, reduces inflammation, and long-term use has a lifelong effect (Patel et al., 2010).

Q10: Stimulates electron exchange in mitochondria and protects against oxidative effects, participates in ATP synthesis. Its deficiency causes severe malfunctioning of the mitochondria (Saini, 2011). Statins inhibit synthesis in the body (Langsjoen and Langsjoen, 2003).

Resveratrol: Stimulates mitochondrial formation, enhances the activity of sirtuin genes, protects telomeres, improves carbohydrate metabolism, and prolongs life (De Paepe et al., 2017). However, it is poorly utilized and rapidly decreases in blood levels, and therefore further active pterostil is recommended (Kauffman, 2017).

Glutathione: The most important antioxidant that protects mitochondria from oxidative effects (Marí et al., 2009).

N-Acetylcysteine (NAC): A precursor to glutathione synthesis, and itself a free radical neutralizing agent, prevents the hepatic effects of paracetamol (Mokhtari et al., 2017).

(R)-Alpha-Lipoic Acid: Mitochondrial nutrient, coenzyme, protects against oxidative effects, reactivates oxidized vitamin C and glutathione, enhances mitochondrial respiration, improves memory and is effective in neuropathy (Liu, 2008).

Astaxanthin: Protects mitochondria from oxidative effects (Wolf, 2010).

Creatine: Protects mitochondria and thereby reduces age-related muscle mass loss (Barbieri et al., 2016).

Melatonin: Contrary to popular belief, it is not only produced in the pineal gland and is not only a “sleep” hormone but is also actively involved in protecting the mitochondria from oxidative effects (Kauffman, 2017).